Fig. 8. Maternal llk/scrb1 is required for convergent extension movements and genetically interacts with tri/stbm. (A-F) Maternal and zygotic (MZ-) llk^rw468 embryos show slight convergent extension (CE) defects. Wild-type (A,D), MZ-llk^rw468 (B,E) and scrb1 MO/ATG-injected (C,F) embryos were observed when alive (A-C) or labeled with myoD RNA probe (Weinberg et al., 1996) (D-F). In MZ-llk^rw468 and scrb1 MO/ATG-injected embryos, the anterior-posterior axis was shorter and somatic mesoderm wider than wild-type embryos. (G-J) Morphology of embryos recovered in the later stages; only tail regions are deficient in MZ-llk^rw468 embryos (H,J; compare with wild-type embryos shown in G,I). (K-N) llk/scrb1 genetically interacts with tri/stbm. (K) Wild-type embryos injected with stbm MO show slight CE defects. (L) MZ-llk^rw468 embryos injected with stbm MO had slightly greater CE defects. (M) Wild-type embryos injected with scrb1 mRNA had slight CE defects. (N) Wild-type embryos co-injected with stbm MO and scrb1 mRNA showed severe CE defects. (K-L) Images of the nVII motor neurons in each embryo are shown in insets. Scale bars: 100 µm.