Fig. 1. pol pll1 mutant tissue phenocopies wus mutants. (A,B) pol pll1 seedlings were not viable because of major defects in the basal embryo and vascular development compared with wild-type siblings (C,D). (B,D) Higher magnification of A and C, respectively. (E) pol pll1 apical tissue can be rescued by grafting to the hypocotyl and root of a wild-type seedling. (F-T) Grafted pol pll1 in the ER⁺ (F,K,P), er^- (G,L,Q) and clv3-2 (H,M,R) backgrounds exhibited meristem termination similar to wus-1 mutants (I,N,S) during vegetative (F-J), inflorescence (K-O) and flower (P-T) development. (J,O,T) Wild-type Ler shown as a control. Similar meristem termination, albeit with reduced penetrance and expressivity, was observed in pol/pol pll1/+ plants (U,V), pol mutants expressing antisense PLL1 (W,X), and pol pll1 mutants incompletely rescued by P_PLL1:PLL1 expression (Y). Arrows indicate junction of cotyledon vascular elements (B,D) or graft junction (E). Asterisks indicate flowers that developed with reduced or absent gynoecia. Insets in V and X show flowers with reduced or absent gynoecia, respectively, at higher magnifications.