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Figure 3


Fig. 3. nmo antagonizes BMP signaling during Drosophila wing development. (A) dpp-Gal4>UAS-tkvQD results in a bifurcated wing blade. (B) dpp-Gal4>UAS-nmo has no visible wing defect. (C) Ectopic nmo is able to suppress the bifurcated phenotype in UAS-nmo/+; dppGal4/UAS-tkvQD wings. (D) ptc-Gal4>UAS-tkv causes loss of wing tissue and fusion of L3 and L4 veins. (E) ptc-Gal4>UAS-nmo shows no obvious phenotype. (F) ptc-Gal4/UAS-nmo; UAS-tkv/+ shows suppression of the ectopic tkv phenotype. (G) vg-Gal4>UAS-Mad showing both a widened wing blade and ectopic veins. (H) vg-Gal4>UAS-nmo shows no obvious phenotype. (I) UAS-Mad/+; vg-Gal4/UAS-nmo rescues the broad wing blade and ectopic wing veins phenotype caused by ectopic Mad. (J) The weak Dad mutant Dadj1E4 has no discernible wing phenotype. (K) nmoadk1 showing a mild ectopic vein phenotype. (L) Dadj1E4; nmoadk1 double-mutants have more severe ectopic vein phenotypes than nmoadk1 alone.