Fig. 1. opa and dpp interact genetically and produce identical head capsule defects. Scanning electron micrographs of wild-type (A) and head-capsule defect heads in opa and/or dpp mutants (B-G). (B) dpp^TgR46.1/Df(2L)DTD2, P20 (a strong dpp head capsule mutant phenotype), (C) opa^12.3/+; dpp^TgR46.1/+ and (D) opa^ts125/opa^12.3. (E) Missing palpus and disordered vibrissae from opa^12.3/+; dpp^TgR46.1/+. (F) Enlargement of the palpus in C. (G) Enlargement of vibrissae region in D. dpp^s-hc homozygotes, dpp/+;opa/+, and opa^ts125/opa^12.3 have identical head capsule defects; eyes are smaller and rounder than control (compare the length of brackets), vibrissae are disrupted and clustered together (arrows), the maxillary palps are altered in shape and in number (circles).