Fig. 5. Genetic interaction of PGPs with RCN1. (A-D) Aberrant cell divisions of the hypophysis at the globular stage in rcn1pgp1pgp19 mutant embryos. The wild-type hypophysis divides into two derivatives: the smaller lens-shaped cells and bigger basal cells (A). Different aberrations in the cell division of the rcn1pgp1pgp19 mutant (B-D). (E,F) Rootless (E) and cotyledon patterning (F) defects in rcn1pgp1pgp19 seedlings (n=97). (G) Enhanced defects in root elongation and gravitropism in 10-day-old seedlings of rcn1pgp1pgp19 as compared with controls. (H,I) Defects in root tip organization, visualized by a lugol staining in rcn1pgp1pgp19 (I) when compared with wild type (H). (J) Quantification of root length and gravitropism phenotypes of the rcn1pgp1pgp19 mutant. For comparison, pin2 and pin2pgp1pgp19 data are also included (n=25). Scale bars: 2 mm. Error bars represent s.e.m.