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First published online January 13, 2009
doi: 10.1242/10.1242/dev.031682

Development 136, 349-353 (2009)
Published by The Company of Biologists 2009
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Triggering the regeneration and tissue repair programs

Elly Tanaka1

1 Center for Regenerative Therapies in Dresden (CRTD), D-01307 Dresden, Germany.
2 Department of Zoology and Animal Biology, University of Geneva, CH-1211 Geneva 4, Switzerland.

Brigitte Galliot2


Figure 1
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  		Fig. 1. Hemocytes recruited to an epithelial wound in the epithelium of a Drosophila embryo. Hemocytes are in red (false-colored), epithelium in green (GFP-moesin). Image courtesy of Brian Stramer and Paul Martin.

 

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  		Fig. 2. Parallels between the signaling pathways of Prod1 and the urokinase plasminogen activator receptor. Prod1 is a salamander three-finger protein with a GPI anchor and a secreted protein ligand, nAG. The mammalian urokinase plasminogen activator receptor (PLAUR; uPAR) is also GPI-linked and has 3 three-finger domains. Both proteins can activate the MAP kinase pathway (ERK/MAPK) by interacting with the EGF receptor, and can activate transcription of the matrix metalloprotease MMP9, which is an early player in limb regeneration. Putative interactions are indicated by question marks. Image courtesy of Jeremy Brockes.

 

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© The Company of Biologists Ltd 2009