Fig. 4. Activating the EGFR pathway rescues the oenocyte deficit in abdA mutants. (A,B) AbdA driven by en-GAL4 (A) or ato-GAL4 (B) rescues oenocyte formation or Rho maintenance respectively in abdA^M1 homozygotes. (C-E) sSpi driven by ato-GAL4 (C) or en-GAL4 (D) in an abdA^M1 background rescues oenocyte formation. In addition, anti-Futsch/22C10 labelling reveals that a dorsal or lateral array of 5-7 chordotonal organs (arrowheads) is produced with en-GAL4 (E), instead of the dorsal triplet found in abdA mutants (Heuer and Kaufman, 1992). (F) The lack of oenocytes in ato¹/Df(3R)p13 transheterozygotes (Elstob et al., 2001), is rescued by driving Rho with en-GAL4. (G) Providing EGFR^ACT with sal-GAL4 in an abdA^M1 background produces a normal oenocyte pattern. (H) Numbers of oenocytes per abdominal cluster produced with the GAL4-driver/UAS combinations indicated.

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