Fig. 1. Components of the Bmp pathway interact genetically with integrins. (A) A wild-type wing. Longitudinal L2, L3, L4 and L5 veins, and anterior (acv) and posterior (pcv) crossveins are indicated. Anterior is towards the top, and proximal towards the left. (B) As previously reported, certain allelic combinations of myospheroid, such as mys¹/mys^nj42, produce wings with ectopic vein material (asterisks) or vein thickening (arrow). Escapers from mys^XR04/mys^nj42 exhibit both ectopic veins and vein truncations (not shown). (C) The enhancer piracy-sog line sog^EP7 induces small distal truncations of the L4 vein (arrow). Asterisk indicates ectopic vein material. (D) The mys¹ null allele enhances the sog^EP7 truncation phenotype (arrows), while the mew^M6 null allele suppresses this phenotype (E). Note that the mys¹ and mew^M6 alleles both enhance the amount of ectopic vein material between L2/L3 (asterisk). (F) A scb² null allele also enhances vein truncation; however, no ectopic veins are seen between L2/L3. (G) Thickened veins are produced in tkv¹ mutants (arrows). This phenotype is suppressed by the ßPS allele mys^nj42 (H).

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