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Fig. 5. Embryonic phenotype of a subset of genes from the screen reveal specific morphological defects. Lateral views (A-C) of late stage 16 embryos of the genotypes wild-type (A), ast¹/Df(2R)ast4 (B) and trn^25.4/In(3LR)C190 (C) stained with an anti-Mhc antibody (see Materials and methods for details). The same symbols are used in both A and B to designate the same muscles. A diagramatic representation of visible muscles is included within each panel, with missing muscles colored red and muscles showing morphological abnormalities colored blue. Loss of ast (B) function leads to muscle-specific losses, as exemplified by loss of muscle LL1 and DO4. Mutations in trn (C) specifically blocked normal muscle morphogenesis, without interfering with the initial specification. This is illustrated by two complete sets of lateral transversal muscles showing aberrant morphologies (1 to 4 indicate LT1-4, and 5 indicates DT1).

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