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Fig. 8. XRAR{alpha}2.2 loss-of-function alters the expression of FGF8, FGFR4 and FGFR1. Embryos were microinjected unilaterally with ß-galactosidase mRNA plus 10 ng RAR-MO or 10 ng RAR-MO plus 1 ng XRAR{alpha}2 mRNA. Embryos were fixed when control uninjected embryos reached stage 18, stained for ß-galactosidase activity and then processed for in situ hybridization with FGF8 (A-F), FGFR4 (G-L) or FGR1 (M-R) probes.





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