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Fig. 7. Apical patterning during embryogenesis. (A-D) Mutant phenotypes. (A,B)
Apical region of the early-heart stage embryo. (A) Wild-type apex is divided
into three subregions, presumptive SAM (PS; orange), cotyledon primordia (CP;
yellow) and boundary of cotyledon margins (BCM; blue). (B) In the
pin1-3 or pin1-3 pid-2 embryo, the peripheral region
possesses a mixed identity of CP and BCM, which is shown in green. (C,D) Late
heart stage. The area of BCM is reduced in pin1-3 (C), while the
entire peripheral region continues to express a mixed identity of CP and BCM
in pin1-3 pid-2 (D). (E,F) Model for apical patterning during
embryogenesis. (E) PID expression (blue) accumulates mainly in the
boundaries of cotyledon primordia and slightly in regions that surround the
base of cotyledon primordia. PIN1 and PID redundantly
promote auxin transport toward the tips of the cotyledon primordia (brown
arrows), resulting in the formation of auxin gradient maxima (green). (F)
Auxin accumulation in cotyledon primordia (green) prevents CUC gene expression
(yellow) from expanding to the periphery.
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