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Fig. 8. Fak56 and ßmys-integrin interact genetically in the Drosophila wing. Wing blister formation induced by expression of Fak56 in the developing wing disc is sensitized in an integrin mutant background. (A) Light micrographs of wings overexpressing Fak56. A(i) Engrailed-GAL4/+ control showing wild-type wing morphology. Wing veins L1-5, anterior cross vein (ACV) and posterior wing vein (PCV) are indicated. A(ii) Engrailed-GAL4/+;UAS-Fak56/+ male, A(iii) ßmysb43;Engrailed-GAL4/+;UAS-Fak56/+ male wings are shown. Arrows indicate wing defects or blisters. (B) Quantification of the wing blistering observed for the genotypes: Engrailed-GAL4/+;UAS-Fak56/+ female (n=241) and male (n=172), ßmysnj42;Engrailed-GAL4/+;UAS-Fak56/+ female (n=246) and male (n=117) and ßmysb43;Engrailed-GAL4/+;UAS-Fak56/+ female (n=160) and male (n=154) is shown. Numbers in brackets after the genotype indicate the number of flies scored.





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