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Fig. 8. The mrp-1 gene acts neither in the cGMP nor in the TGF-ß
signaling pathway for dauer larva regulation. (A) The mrp-1(pk89)
mutation enhanced the dauer larva formation of daf-11(m47) (cGMP
signaling pathway) and daf-1(m40) (TGF-ß signaling pathway) to a
small and similar extent. By contrast, it enhanced that of
daf-2(e1370ts) (insulin signaling pathway) to a large extent. (B) The
dauer larva formation of daf-2(e1370);mrp-1(pk89) was not suppressed
by che-3(e1124), a daf-d mutation in the cGMP signaling
pathway. (C) The dauer larva formation of daf-2(e1370);mrp-1(pk89)
was not suppressed by daf-5(e1386), a daf-d mutation in the
TGF-ß signaling pathway. (D) As expected, the dauer larva formation of
daf-2(e1370);mrp-1(pk89) was suppressed by daf-16(mu86), a
daf-d mutation in the insulin signaling pathway, which suppresses
daf-2(e1370).
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