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Fig. 3. nmo antagonizes BMP signaling during Drosophila wing development. (A) dpp-Gal4>UAS-tkv^QD results in a bifurcated wing blade. (B) dpp-Gal4>UAS-nmo has no visible wing defect. (C) Ectopic nmo is able to suppress the bifurcated phenotype in UAS-nmo/+; dppGal4/UAS-tkv^QD wings. (D) ptc-Gal4>UAS-tkv causes loss of wing tissue and fusion of L3 and L4 veins. (E) ptc-Gal4>UAS-nmo shows no obvious phenotype. (F) ptc-Gal4/UAS-nmo; UAS-tkv/+ shows suppression of the ectopic tkv phenotype. (G) vg-Gal4>UAS-Mad showing both a widened wing blade and ectopic veins. (H) vg-Gal4>UAS-nmo shows no obvious phenotype. (I) UAS-Mad/+; vg-Gal4/UAS-nmo rescues the broad wing blade and ectopic wing veins phenotype caused by ectopic Mad. (J) The weak Dad mutant Dad^j1E4 has no discernible wing phenotype. (K) nmo^adk1 showing a mild ectopic vein phenotype. (L) Dad^j1E4; nmo^adk1 double-mutants have more severe ectopic vein phenotypes than nmo^adk1 alone.

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