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Fig. 8. The dPSR-induced genital rotation defect is suppressed through the JNK
pathway in Drosophila. (A) The penetrance of the
rotation defect caused by expression of dPSR was suppressed by removing one
copy of puc observed in w; UAS-dPSR/act5c-GAL4 or w;
UAS-dPSR/act5c-GAL4; pucE69lacZ/+ flies (observed percentage
of 642 and 496 males analyzed, respectively). (B) The pathway through
which dPSR may act to inhibit cell death.