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Fig. 1. drn mutant and drn drnl double-mutant phenotypes.
Wildtype embryos at early (A) and late (B) globular stages.
Abnormal hypophyseal cell divisions in drn-1 mutant embryos at early
(C) and late (D) globular stages. (E) Multiple cell files
in the suspensor of a drn1 mutant; the embryo proper is towards the
top. (F) drn-1 mutants lacking basal embryo organisation.
(G-K) Pleiotropy of cotyledon phenotypes for the drn-1 mutant
showing mono-(G) and polycotyledony (I), cotyledon fusion (H) and cup-shaped
cotyledons (J). The SAM is active despite complete cotyledon fusion (K).
(L,M) mp-like phenotype of drn (L) and
drn-1 drnl-1 (M) mutants. (N,O) Altered phyllotaxis of
first leaves in monocotyledonous (N) or polycotyledonous (O) drn-1
mutants. (P-R) Cleared cotyledons of wild type (P) and a
monocotyledonous drn-1 mutant showing single (Q) or fused cotyledon
(R) ontogeny. (S,T) drn-1 drnl-1 double-mutant embryos
showing asymmetric cell divisions at early (S) or late (T) globular stages.
(U,V) pin-like hypocotyl of drn-1 drnl-2
double-mutant plant lacking cotyledons (U) and directly producing leaves as
evidenced by trichome formation (V).
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