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Fig. 5. Genetic interaction of PGPs with RCN1. (A-D) Aberrant cell
divisions of the hypophysis at the globular stage in rcn1pgp1pgp19
mutant embryos. The wild-type hypophysis divides into two derivatives: the
smaller lens-shaped cells and bigger basal cells (A). Different aberrations in
the cell division of the rcn1pgp1pgp19 mutant (B-D).
(E,F) Rootless (E) and cotyledon patterning (F) defects in
rcn1pgp1pgp19 seedlings (n=97). (G) Enhanced defects
in root elongation and gravitropism in 10-day-old seedlings of
rcn1pgp1pgp19 as compared with controls. (H,I) Defects
in root tip organization, visualized by a lugol staining in
rcn1pgp1pgp19 (I) when compared with wild type (H). (J)
Quantification of root length and gravitropism phenotypes of the
rcn1pgp1pgp19 mutant. For comparison, pin2 and
pin2pgp1pgp19 data are also included (n=25). Scale bars: 2
mm. Error bars represent s.e.m.
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