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Fig. 5 CA babo genetically interacts with the components of the Rho GTPase pathway. (A) Quantification of CA Babo defects in the presence of control (w¹¹¹⁸) or one mutant copy of Rho or Smad, as indicated. CA Babo phenotypes were classed according to the loss or truncation of dorsal (D^-M⁺), medial (D⁺M^-) or both (D^-M^-) lobes. Axon fasciculation defects were also observed (classed as misguidance, MG; see Fig. S1 in the supplementary material). Based on the level of Babo expression (see Materials and methods), misguidance represents the strongest phenotype and loss of dorsal lobes the weakest phenotype (MG > D⁺M^- > D^-M^- > D^-M⁺). The asterisk denotes CA Babo-induced β lobe overextension upon the loss of one copy of LIMK1. (B) Quantification of CA Babo defects in control (UAS-mCD8::GFP), or with one copy of the indicated transgene. n, number of hemispheres examined.

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