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Figure 2


Fig. 2. Increasing the levels of Hipk can compensate for the loss of the Wg signaling. (A) Wild-type wing. (B) UAS-hipk/69B-Gal4. (C,D) Misexpression of hipk produced a mild ectopic vein phenotype similar to what is seen with UAS-armS10/+; bs-gal4/+ (C) or nmoDB24/nmoadk(D). (E) The hypomorphic wg allelic combination wg1/wg1-17 caused a wing-to-notum transformation, which was rescued by co-expression of hipk (F). (G) UAS-DFz2N33/+; 69B-Gal4/+ causes a severely notched wings. (H) UAS-DFz2N33/+; 69B/UAS-hipk. Simultaneously misexpressing hipk rescued the loss of wing tissue caused by ectopic expression of DFz3N33. (I) UAS-DFz2N33/+; dpp-Gal4/+. (J) UAS-DFz2N33/+; dpp-Gal4/hipk4. (K) sd>DAxin causes mild notches and loss of posterior margin bristles that are enhanced by loss of one copy of hipk in 32B>Daxin, hipk3/+ (L).





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